Cell Survival And Cell Death (Cold Spring Harbor Perspectives In Biology)


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Bookseller Inventory # GB-9781936113316 More Information About This Seller Ask Bookseller a Question Buy New US$ 101.90 Convert Currency Add to Basket Shipping: US$ 13.03 From United Kingdom to U.S.A. Distinct death mechanisms in Drosophila development. Cite/Export Cite/Export Copy a citation APA (6th ed.) Chicago (Author-Date, 15th ed.) Harvard (18th ed.) MLA (7th ed.) Turabian (6th ed.) Export a citation Export to RefWorks Export to EndNote / Reference Manager Export to EasyBib Export to EndNote / Reference Manager(non-Latin) Cancel Note: Citations are based on reference standards. Green – Mechanisms of action of Bcl-2 family proteins / Aisha Shamas-Din, Justin Kale, Brian Leber, and David W. User Name: Password: Remember me on this computer Cancel Forgot your password? . Cold Spring Harb Perspect Biol. Tissue-specific gene expression and ecdysone-regulated genomic networks in Drosophila. Class III PI3K Vps34 plays an essential role in autophagy and in heart and liver function. Cancer Biol Ther 8: 20842096 [PMC free article] [PubMed] Mathew R, Karp CM, Beaudoin B, Vuong N, Chen G, Chen HY, Bray K, Reddy A, Bhanot G, Gelinas C, et al.

Annu Rev Biochem 80: 125–156 [PubMed] Wullschleger S, Loewith R, Hall MN 2006. 2003). Interestingly, midgut destruction is blocked in animals with impaired Atg1, Atg2, or Atg18 function, directly implicating autophagy as a crucial process in steroid-induced degradation of midgut cells (Denton et al. Apoptosis and other cell death mechanisms are complex and carefully controlled. Billions of cells die every day in the human body. Freel, and Sally Kornbluth – Caspase substrates and inhibitors / Marcin Poreba, Aleksandra Strozyk, Guy S. Steroid regulated programmed cell death during Drosophila metamorphosis. Cancer Cell 10: 51–64 [PMC free article] [PubMed] Denton D, Shravage B, Simin R, Mills K, Berry DL, Baehrecke EH, Kumar S 2009. Treatment of MCF-7 mammary cancer cells that lack caspase-3 with 4-hydroxytamoxifen triggers cell death with autophagy, suggesting the possibility that autophagy can compensate for defects in apoptosis (Bursch et al.

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